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Deficiency of Capicua disrupts bile acid homeostasis
Eunjeong Kim
, Sungjun Park
, Nahyun Choi
, Jieon Lee
, Jeehyun Yoe
, Soeun Kim
, Hoe Yune Jung
, Kyong Tai Kim
, Hyojin Kang
, John D. Fryer
, Huda Y. Zoghbi
, Daehee Hwang
, Yoontae Lee
Department of Biology
KNU Creative BioResearch Group
Project_LAMP Group
Pohang University of Science and Technology
National Institute of Supercomputing and Networking
Mayo Clinic College of Medicine and Science
Texas Children's Hospital Houston
Institute for Basic Science
Research output
:
Contribution to journal
›
Article
›
peer-review
29
Scopus citations
Overview
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Biochemistry, Genetics and Molecular Biology
Homeostasis
100%
Bile Acid
100%
Mouse
66%
Proinflammatory Cytokine
33%
FOXA2
33%
CCAAT Enhancer Binding Protein Beta
33%
Drug Metabolism
16%
Metabolic Disorder
16%
Transporter Genes
16%
Nuclear Factor I
16%
Hepatocyte Nuclear Factor 3beta
16%
Tumor Necrosis Factor
16%
Immunology and Microbiology
Mouse
100%
Homeostasis
100%
Proinflammatory Cytokine
50%
CCAAT Enhancer Binding Protein Beta
50%
Tumor Necrosis Factor Alpha
25%
Gallbladder
25%
Hepatocyte Nuclear Factor 3beta
25%
Pharmacology, Toxicology and Pharmaceutical Science
Bile Acid
100%
Mouse
66%
Cytokine
33%
Malignant Neoplasm
16%
Chronic Liver Disease
16%
Metabolic Disorder
16%
Tumor Necrosis Factor
16%
Spinocerebellar Degeneration
16%
Hepatocyte Nuclear Factor 3beta
16%
Inflammation
16%
Drug Metabolism
16%
Cholestasis
16%
Neuroscience
Bile Acid
100%
Proinflammatory Cytokine
33%
Retinoid X Receptor Alpha
33%
FOXA2
33%
In Vivo
16%
Tumor Necrosis Factor Alpha
16%
Metabolic Disorder
16%
Nuclear Factor I
16%
Hepatocyte Nuclear Factor 3beta
16%
Metabolic Pathway
16%