Abstract
The resistance mechanism of extended-spectrum cephalosporins in clinical isolates of Citrobacter freundii, Enterobacter spp., and Serratia marcescens was studied. Of 152 isolates, 45 isolates (29.6%) were derepressed AmpC mutants and 39 isolates (25.7%) produced extended-spectrum β-lactamase (ESBLs). The most prevalent ESBLs were CTX-M enzymes, followed by TEM-52 and SHV-12.
| Original language | English |
|---|---|
| Pages (from-to) | 2452-2455 |
| Number of pages | 4 |
| Journal | Journal of Clinical Microbiology |
| Volume | 43 |
| Issue number | 5 |
| DOIs | |
| State | Published - May 2005 |
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